This conclusion agrees with that of MIT's. According to the analysis to the RBCs'structure, components and their physiological functions, double exponential analysis to autocorrelation function of erythrocyte and ghost yields Г〓 and Г〓.
根据对红细胞的生理功能和结构成分分析，使用双指数分析得到〓和〓分别描述红细胞内Hb分子的热扩散运动和细胞膜表面的起伏波动。
The results show that MQLS results of normal RBC differ markedly from that of ghost (p.05). Both autocorrelation function curves of erythrocytes and ghosts show a double peak distribution, which means that both Brown motions of intracellular Hb and fluctuations of membranes contribute to autocorrelation function of erythrocyte and ghost. The contribution from cell skeleton is almost zero.
测量结果还表明红细胞与血影细胞的MQLS明显不同（p.05），红细胞与血影细胞的ACF曲线均呈双峰分布，提示红细胞的动态光散射来源主要有两个，红细胞内Hb分子的Brown运动和红细胞膜上的涨落都对红细胞ACF有贡献，而红细胞骨架的贡献几乎为O，这与MIT的结论相同。
The extent of fusion for human erythrocyte ghost membrane and PS liposome increases with increasing the membrane fluidity within a certain range, and decreases with increasing the membrane fluidity beyond this range.
血影膜、PS脂质体的最大融合程度在一定范围内随膜流动性增加而增大，超出此范围则随膜流动增加而减小。
METHODS:The human erythrocyte white ghost were pre-treated by 100 mmol/L NaOH and then digested by trypsin. Using the HPLC and amino acid sequencer, we identified the peptides that released from white ghost, and purified the C1 peptides(band 3 Ala893-Va1911). The C1 peptides were used to incubate with fresh white ghosts.
以100 mmol/L NaOH预处理红细胞膜血影，再用胰蛋白酶消化，采用高效液相色谱及氨基酸测序仪分析检定了从红细胞血影中释放的多肽，并纯化了带Ⅲ蛋白C1肽链，以不同浓度的C1肽链与新鲜的红细胞血影进行温浴。
Calcium phosphate-induced erythrocyte ghost membrane fusion and PS lipo-some fusion at a certain temperature were studied by Tb/DPA fluorescent technique.
复旦大学生物物理教研室，上海；天津市中国医学科学院生物医学工程研究所，天津
To establish a model of oxidative damage in erythrocyte ghost induced by superoxide anion free radicals (SAFR), and to study the protective action of portulaca total flavone on oxidative damage was induced by SAFR which was produced by auto-oxidation of pyrogallic acid.
为建立超氧阴离子自由基（O2·-）引发人红细胞膜氧化损伤实验模型，研究马齿苋总黄酮对红细胞膜氧化损伤的保护作用。
Result O · _2 caused lipid peroxidation of erythrocyte ghost, increased the content of MDA and the speed of auto-oxidation, decreased membrane fluidity, and alleviated blocking degree.
结果O2.能引起红细胞膜脂质过氧化，丙二醛（MDA）含量、自氧化速率增加、膜流动性以及膜封闭度下降。
The results showed that propolis could significantly inhibit lipid oxidation of rat erythrocyte ghost induced by action oxygen free radicals (peroxy radical and hydroxy radical) with dose-dependent effect.
结果表明，蜂胶能显著地抑制氧自由基引发的鼠红细胞膜脂质过氧化反应，脂质过氧化产物丙二醛（MDA）含量显著下降，呈现量效关系。
Diphenyl 1, 3, 5 hexatriene, a kind of fluorescence probe, was used to observe the effects of tea pollen flavonoids on the oxidative damage in rat erythrocyte ghost induced by peroxy radical and hydroxy radical.
应用1,6-二苯基-1,3,5-已三烯（DPH）为荧光探针，超氧阴离子自由基和羟基自由基致鼠红细胞膜氧化损伤为模型，研究了茶叶花粉黄酮对鼠红细胞膜氧化损伤的影响。
Methods Human DAF was purified from human erythrocyte ghost by trypsin digestion, butanol extraction, sequential chromatography on DE32 and immunoaffinity chromatography.
方法经胰蛋白酶消化、正丁醇抽提及DE32离子交换色谱和抗体亲和层析等步骤从人红细胞膜影中分离纯化人红细胞膜DAF。